Ciproton is a medication used for the treatment of stomach ulcers, short-term treatment of erosive esophagitis due to gastroesophageal reflux disease (GERD), maintenance of healing of erosive esophagitis, and pathological hypersecretory conditions including Zollinger–Ellison syndrome. It may also be used along with other medications to eliminate Helicobacter pylori. Effectiveness is similar to other proton pump inhibitors (PPIs). It is available by mouth and by injection into a vein.
Other names for this medication:
Pantoprazole,
Protonix,
Aciban,
Acipan,
Anagastra,
Anesteloc,
Apazol,
Apton,
Caprol,
Ciproton,
Contix,
Contracid,
Controloc,
Digene,
Eupantol,
Gastromax,
Gastrowell,
Noacid,
Nolpaza,
Ozepran,
Pandev,
Pandon,
Panpac,
Pansec,
Pantac,
Pantaz,
Pantecta,
Pantin,
Pantium,
Pantoc,
Pantocal,
Pantocalm,
Pantocid,
Pantodac,
Pantor,
Pantorc,
Pantosec,
Pantozol,
Pantpas,
Pantul,
Pantus,
Panum,
Panzo,
Panzol,
Pentalink,
Pents,
Peptac,
Peptazol,
Pepzol,
Prazolan,
Progen,
Tecta,
Tonval,
Topra,
Topraz,
Topzole,
Trupan,
Ulcemex,
Ulcepraz,
Ulcoreks,
Ulcotenal,
Unigastrozol,
Zacpac,
Zepoxin,
Zimpax,
Zipant,
Zipantola,
Ziprol,
Zovanta,
Zurcal,
Zurcazol,
Similar Products:
Bapex,
Bexinor,
Uzol,
Cizole,
Emidoxyn,
Norfloxacin,
Virol,
Losepine,
Novamin,
Vetrimoxin,
Zorax,
Amoklavin,
Imda,
Prazolit,
Terasin,
Zenil,
Celtium,
Lutamidal,
Stromectol,
Monamox,
Candistat,
Sensival,
Metris,
Torgyn,
Biodasin,
Bupropion,
Dolmax,
Amoxicillinum,
Attera,
Omexel,
Alpentin,
Ocid,
Anazol,
Ritonavir,
Rebetron,
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Tissue plasminogen activator (tPA) mediates a number of processes that are pivotal for synaptogenesis and remodeling of synapses, including proteolysis of the brain extracellular matrix, degradation of adhesion molecules, activation of neurotrophins, and dejac tablet repair activation of the N-methyl-d-aspartate receptor. A systematic literature research was performed in the Pubmed and Medline database up to August 2007. Increased galectin-3 expression and secretion is a feature of alternative macrophage activation. Individuals with Down syndrome (DS) are at an increased risk for certain maladaptive behaviors. There has been much speculation that the interaction of the two mechanisms and the resulting fine structure limits the clinical test performance of DPOAEs.