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Alzen (Quetiapine)
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Quetiapine

Alzen works through two mechanisms. The first is by acting on dopamine receptors. Dopamine is a signaling molecule in the brain that plays an important role in motor control and thinking abilities. In Huntington’s disease patients, dopamine levels are imbalanced. In the early and intermediate stages of the disease, dopamine’s activity is too high, which is thought to cause chorea. Alzen binds to dopamine receptors, preventing dopamine itself from binding to its receptor, thereby interfering with its function. The second mechanism through which Alzen acts is by blocking serotonin receptors, primarily one called 5HT2A. Inappropriate binding of serotonin, another signaling molecule in the brain, to the 5HT2A receptor plays a role in psychiatric disorders, such as schizophrenia, dyskinesia (difficulties with voluntary movements), and depression. It is not entirely understood how the combination of these two mechanisms results in the therapeutic effects of Alzen in Huntington’s disease patients.

Other names for this medication:
Quetiapine, Seroquel, Alzen, Asicot, Cedrina, Ketiapin, Ketilept, Ketipinor, Kventiax, Norsic, Quetapel, Quetiapin, Quetiapina, Quetiazic, Quetidin, Quetin, Serenil, Setinin, Socalm, Vesparax,

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Carbon monoxide-related fatalities: A 60-year single institution experience. The deformed defect is theoretically confirmed to be an array of four linearly aligned singly charged vortices. Exercise tests improve the prediction of cardiac death in ischemic heart disease. Finally, the existence of a relationship between the information transfer and the level of brain synchronization as measured by phase synchronization value (PLV) was investigated. Hypothermia can restrain the production of oxygen free radicals following testicular torsion/detorsion in rats, which in turn can inhibit lipid peroxidation and increase the survivability of the torsional testis.

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